In this study, the scientists used the CRISPR system to explore the more translational area between the research in neuroscience and conceptually novel treatments.
Their findings fill the gap of our knowledge regarding the relationship between SCN1A mutation effect recorded on exogenously transfected cells and on Nav1.1-expressing neurons, and reveal the physiological basis underlying epileptogenesis caused by SCN1A loss-of-function mutation.
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In this study, the scientists used the CRISPR system to explore the more translational area between the research in neuroscience and conceptually novel treatments.
Their findings fill the gap of our knowledge regarding the relationship between SCN1A mutation effect recorded on exogenously transfected cells and on Nav1.1-expressing neurons, and reveal the physiological basis underlying epileptogenesis caused by SCN1A loss-of-function mutation.